TCTAP C-038 2-vessel Coronary Artery Disease with Over 300μm Thick Calcification Treated by Cutting Balloon
نویسندگان
چکیده
منابع مشابه
Coronary artery calcification: does it predict obstructive coronary artery disease?
Calcification of the coronary arteries plays a key role in the pathophysiology of atherosclerosis. Coronary calcification is an active process culminating in extracellular matrix deposition of calcium by osteoblast-like cells that has some resemblance to bone formation [1]. Subintimal coronary calcification is almost exclusively associated with the presence of coronary atherosclerosis and is co...
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OBJECTIVE Studies in mice have shown that genetic disruption of monocyte chemotactic protein-1 or its receptor, the C-C chemokine receptor 2 (CCR2), inhibits atherosclerosis, but few data exist in humans to suggest that the monocyte chemotactic protein-1-CCR2 interaction is important in atherogenesis. A common polymorphism in the human CCR2 gene resulting in a substitution of isoleucine for val...
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Introduction: Heart failure is a major hazard for public health. Despite recent advance in medical therapy, there is not enough information on the outcome of off-pump coronary artery bypass (OPCAB) and medical therapy on the patients with severe ventricular dysfunction and triple-vessel (CAD). This study aimed to compare treatment outcomes and mortality rate in patients undergoing off-pump coro...
متن کاملEvaluation of Serum Levels of Mannose Binding Lectin-2, Tenascin-C in Patients with Coronary Artery Disease (CAD)
Cardiovascular disease (CVD) is a leading cause of death worldwide and continues to increase in prevalence compared to previous decades, in part because of the aging of the world population. Atherosclerotic CVD starts at a very young age and progresses over time allowing sufficient time for screening and early detection of the condition. Advances in biomarker research and developments related t...
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ژورنال
عنوان ژورنال: Journal of the American College of Cardiology
سال: 2018
ISSN: 0735-1097
DOI: 10.1016/j.jacc.2018.03.233